An essential role of the NF-kappa B/Toll-like receptor pathway in induction of inflammatory and tissue-repair gene expression by necrotic cells.
第一作者:
M,Li
第一单位:
Department of Biological Sciences, Columbia University, New York, NY 10027, USA.
作者:
主题词
动物(Animals);细胞凋亡(Apoptosis);细胞系(Cell Line);细胞, 培养的(Cells, Cultured);趋化因子CXCL1(Chemokine CXCL1);炎症趋化因子类(Chemokines);趋化因子, CXC(Chemokines, CXC);细胞因子类(Cytokines);果蝇蛋白质类(Drosophila Proteins);胚胎, 哺乳动物(Embryo, Mammalian);成纤维细胞(Fibroblasts);基因表达调控(Gene Expression Regulation);炎症(Inflammation);炎症介导素类(Inflammation Mediators);白细胞介素1(Interleukin-1);白细胞介素8(Interleukin-8);巨噬细胞(Macrophages);膜糖蛋白类(Membrane Glycoproteins);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);NF-κB(NF-kappa B);坏死(Necrosis);受体, 细胞表面(Receptors, Cell Surface);信号传导(Signal Transduction);Toll样受体2(Toll-Like Receptor 2);Toll样受体(Toll-Like Receptors);转录因子RelA(Transcription Factor RelA);伤口愈合(Wound Healing)
DOI
10.4049/jimmunol.166.12.7128
PMID
11390458
发布时间
2022-03-17
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