Supersensitivity to anandamide and enhanced endogenous cannabinoid signaling in mice lacking fatty acid amide hydrolase.
第一作者:
B F,Cravatt
第一单位:
The Skaggs Institute for Chemical Biology and Departments of Cell Biology and Chemistry, The Scripps Research Institute, La Jolla, CA 92037, USA. cravatt@scripps.edu
作者:
主题词
酰胺基水解酶类(Amidohydrolases);动物(Animals);花生四烯酸类(Arachidonic Acids);行为, 动物(Behavior, Animal);脑(Brain);大麻酚类(Cannabinoids);内源性大麻酚类(Endocannabinoids);乙醇胺类(Ethanolamines);小鼠(Mice);小鼠, 基因敲除(Mice, Knockout);痛阈(Pain Threshold);多不饱和脂肪酸酰胺类(Polyunsaturated Alkamides);受体, 大麻酚(Receptors, Cannabinoid);受体, 药物(Receptors, Drug);信号传导(Signal Transduction)
DOI
10.1073/pnas.161191698
PMID
11470906
发布时间
2022-02-24
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