Overexpression of leptin in transgenic mice leads to decreased basal lipolysis, PKA activity, and perilipin levels.
第一作者:
Yaohuang,Ke
第一单位:
Department of Laboratory Medicine, 505 Parnassus Avenue, University of California, San Francisco, CA 94143-0134, USA.
作者:
医学主题词
脂肪组织(Adipose Tissue);衰老(Aging);动物(Animals);体重(Body Weight);载体蛋白质类(Carrier Proteins);环AMP(Cyclic AMP);环AMP依赖性蛋白激酶类(Cyclic AMP-Dependent Protein Kinases);酶激活(Enzyme Activation);脂肪酸类, 非酯化(Fatty Acids, Nonesterified);女(雌)性(Female);基因表达调控(Gene Expression Regulation);甘油(Glycerol);瘦素(Leptin);脂解作用(Lipolysis);男(雄)性(Male);膜蛋白质类(Membrane Proteins);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, 转基因(Mice, Transgenic);磷蛋白类(Phosphoproteins);受体, 肾上腺素能β3(Receptors, Adrenergic, beta-3);受体, 瘦素(Receptors, Leptin);性别因素(Sex Factors);信号传导(Signal Transduction)
DOI
10.1016/j.bbrc.2003.11.053
PMID
14651995
发布时间
2019-06-12
基金项目
DK 46942/DK/NIDDK NIH HHS/United States
HD35142/HD/NICHD NIH HHS/United States
T32-DK07636/DK/NIDDK NIH HHS/United States
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