换一批
换一批Poxvirus protein N1L targets the I-kappaB kinase complex, inhibits signaling to NF-kappaB by the tumor necrosis factor superfamily of receptors, and inhibits NF-kappaB and IRF3 signaling by toll-like receptors.
第一作者：
Gary,DiPerna
第一单位：
Viral Immune Evasion Group, Department of Biochemistry, Trinity College, Dublin 2, Ireland.
作者：
主题词
细胞系(Cell Line);细胞因子类(Cytokines);DNA结合蛋白质类(DNA-Binding Proteins);剂量效应关系, 药物(Dose-Response Relationship, Drug);基因, 报告(Genes, Reporter);遗传载体(Genetic Vectors);人类(Humans);I-κB激酶(I-kappa B Kinase);干扰素调节因子3(Interferon Regulatory Factor-3);白细胞介素1(Interleukin-1);膜糖蛋白类(Membrane Glycoproteins);NF-κB(NF-kappa B);质粒(Plasmids);痘病毒科(Poxviridae);沉淀素试验(Precipitin Tests);蛋白质结合(Protein Binding);蛋白质生物合成(Protein Biosynthesis);蛋白质结构, 三级(Protein Structure, Tertiary);受体, 细胞表面(Receptors, Cell Surface);信号传导(Signal Transduction);Toll样受体(Toll-Like Receptors);转录因子(Transcription Factors);转录, 遗传(Transcription, Genetic);转染(Transfection);肿瘤坏死因子α(Tumor Necrosis Factor-alpha);病毒蛋白质类(Viral Proteins)
DOI
10.1074/jbc.M400567200
PMID
15215253
发布时间
2021-12-03
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