换一批Chronic kidney disease causes defects in signaling through the insulin receptor substrate/phosphatidylinositol 3-kinase/Akt pathway: implications for muscle atrophy.
第一作者：
James L,Bailey
第一单位：
Renal Division, Emory University School of Medicine, WMB 338, 1639 Pierce Drive, Atlanta, GA 30322, USA. james.l.bailey@emory.edu
作者：
医学主题词
动物(Animals);细胞, 培养的(Cells, Cultured);胰岛素(Insulin);胰岛素样生长因子Ⅰ(Insulin-Like Growth Factor I);肾功能衰竭, 慢性(Kidney Failure, Chronic);男(雄)性(Male);肌, 骨骼(Muscle, Skeletal);肌萎缩(Muscular Atrophy);磷酸肌醇3-激酶类(Phosphatidylinositol 3-Kinases);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);大鼠(Rats);大鼠, Sprague-Dawley(Rats, Sprague-Dawley);受体, 胰岛素(Receptor, Insulin);信号传导(Signal Transduction);底物特异性(Substrate Specificity)
DOI
10.1681/ASN.2004100842
PMID
16611720
发布时间
2021-10-20
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