Improved glucose homeostasis in mice with muscle-specific deletion of protein-tyrosine phosphatase 1B.
第一作者:
Mirela,Delibegovic
第一单位:
Cancer Biology Program, Department of Medicine, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts, USA. m.delibegovic@abdn.ac.uk
作者:
主题词
体脂肪率(Adiposity);动物(Animals);体重(Body Weight);肌酸激酶(Creatine Kinase);基因缺失(Gene Deletion);葡萄糖(Glucose);内环境稳定(Homeostasis);胰岛素(Insulin);小鼠(Mice);小鼠, 基因敲除(Mice, Knockout);肌, 骨骼(Muscle, Skeletal);器官特异性(Organ Specificity);蛋白质酪氨酸磷酸酶, 非受体1型(Protein Tyrosine Phosphatase, Non-Receptor Type 1);信号传导(Signal Transduction);噻唑烷二酮类(Thiazolidinediones);甘油三酯类(Triglycerides)
DOI
10.1128/MCB.00959-07
PMID
17724080
发布时间
2018-12-01
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