换一批Autophagy induced by Alexander disease-mutant GFAP accumulation is regulated by p38/MAPK and mTOR signaling pathways.
第一作者：
Guomei,Tang
第一单位：
Department of Pathology, Center for Neurobiology and Behavior, Center for Parkinson's Disease and Other Movement Disorders, Columbia University, New York, NY 10032, USA.
作者：
医学主题词
Alexander病(Alexander Disease);动物(Animals);星形细胞(Astrocytes);自噬(Autophagy);脑(Brain);细胞系, 肿瘤(Cell Line, Tumor);神经胶质原纤维酸性蛋白质(Glial Fibrillary Acidic Protein);人类(Humans);小鼠(Mice);小鼠, 突变型(Mice, Mutant Strains);突变(Mutation);蛋白激酶抑制剂(Protein Kinase Inhibitors);蛋白激酶类(Protein Kinases);RNA, 小分子干扰(RNA, Small Interfering);信号传导(Signal Transduction);TOR丝氨酸-苏氨酸激酶(TOR Serine-Threonine Kinases);空泡(Vacuoles);p38丝裂原活化蛋白激酶类(p38 Mitogen-Activated Protein Kinases)
DOI
10.1093/hmg/ddn042
PMID
18276609
发布时间
2021-12-03
- 浏览27
Human molecular genetics
1540-55页
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