High glucose regulates cyclin D1/E of human mesenchymal stem cells through TGF-beta1 expression via Ca2+/PKC/MAPKs and PI3K/Akt/mTOR signal pathways.
第一作者:
Jung Min,Ryu
第一单位:
Department of Veterinary Physiology, Biotherapy Human Resources Center (BK 21), College of Veterinary Medicine, Chonnam National University, Gwangju, Korea.
作者:
医学主题词
钙信号(Calcium Signaling);细胞周期(Cell Cycle);细胞增殖(Cell Proliferation);细胞, 培养的(Cells, Cultured);细胞周期蛋白D1(Cyclin D1);细胞周期蛋白E(Cyclin E);DNA复制(DNA Replication);细胞外信号调节MAP激酶类(Extracellular Signal-Regulated MAP Kinases);胎血(Fetal Blood);葡萄糖(Glucose);人类(Humans);细胞内信号肽和蛋白质类(Intracellular Signaling Peptides and Proteins);间质干细胞(Mesenchymal Stem Cells);丝裂原激活蛋白激酶类(Mitogen-Activated Protein Kinases);磷酸肌醇3-激酶类(Phosphatidylinositol 3-Kinases);磷酰化(Phosphorylation);蛋白激酶C(Protein Kinase C);蛋白激酶抑制剂(Protein Kinase Inhibitors);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);RNA干扰(RNA Interference);RNA, 信使(RNA, Messenger);重组蛋白质类(Recombinant Proteins);TOR丝氨酸-苏氨酸激酶(TOR Serine-Threonine Kinases);时间因素(Time Factors);转化生长因子β1(Transforming Growth Factor beta1);p38丝裂原活化蛋白激酶类(p38 Mitogen-Activated Protein Kinases)
DOI
10.1002/jcp.22091
PMID
20232305
发布时间
2021-12-03
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