换一批
换一批Activation of Trpv4 reduces the hyperproliferative phenotype of cystic cholangiocytes from an animal model of ARPKD.
第一作者：
Sergio A,Gradilone
第一单位：
Miles and Shirley Fiterman Center for Digestive Diseases, Division of Gastroenterology and Hepatology, Mayo Clinic College of Medicine, Rochester, Minnesota, USA.
作者：
主题词
动物(Animals);胆管(Bile Ducts);钙(Calcium);细胞增殖(Cell Proliferation);疾病模型, 动物(Disease Models, Animal);细胞外信号调节MAP激酶类(Extracellular Signal-Regulated MAP Kinases);人类(Humans);亮氨酸(Leucine);表型(Phenotype);佛波醇酯类(Phorbol Esters);多囊肾, 常染色体隐性(Polycystic Kidney, Autosomal Recessive);原癌基因蛋白质B-raf(Proto-Oncogene Proteins B-raf);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);大鼠(Rats);大鼠, Sprague-Dawley(Rats, Sprague-Dawley);信号传导(Signal Transduction);磺胺类(Sulfonamides);TRPV阳离子通道(TRPV Cation Channels)
DOI
10.1053/j.gastro.2010.04.010
PMID
20399209
发布时间
2021-10-20
- 浏览14
Gastroenterology
304-14.e2页
相似文献
- 中文期刊
- 外文期刊
- 学位论文
- 会议论文
