Melatonin protects against isoproterenol-induced alterations in cardiac mitochondrial energy-metabolizing enzymes, apoptotic proteins, and assists in complete recovery from myocardial injury in rats.
第一作者:
Debasri,Mukherjee
第一单位:
Oxidative Stress and Free Radical Biology Laboratory, Department of Physiology, University College of Science and Technology, University of Calcutta, Kolkata, India.
作者:
主题词
动物(Animals);抗氧化剂(Antioxidants);细胞凋亡(Apoptosis);天冬氨酸氨基转移酶类(Aspartate Aminotransferases);谷胱甘肽(Glutathione);同工酶类(Isoenzymes);异丙肾上腺素(Isoproterenol);L-乳酸脱氢酶(L-Lactate Dehydrogenase);脂质过氧化作用(Lipid Peroxidation);过氧化脂质类(Lipid Peroxides);男(雄)性(Male);褪黑激素(Melatonin);线粒体, 心脏(Mitochondria, Heart);心肌缺血(Myocardial Ischemia);心肌(Myocardium);大鼠(Rats);大鼠, Sprague-Dawley(Rats, Sprague-Dawley)
DOI
10.1111/j.1600-079x.2012.00984.x
PMID
23050266
发布时间
2019-09-23
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Journal of pineal research
166-79页
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