Loss of Wnt5a disrupts second heart field cell deployment and may contribute to OFT malformations in DiGeorge syndrome.
第一作者:
Tanvi,Sinha
第一单位:
Department of Cell, Developmental and Integrative Biology, University of Alabama at Birmingham, Alabama, USA.
作者:
主题词
动物(Animals);鸡胚(Chick Embryo);DiGeorge综合征(DiGeorge Syndrome);胚胎干细胞(Embryonic Stem Cells);基因缺失(Gene Deletion);心脏(Heart);小鼠(Mice);小鼠, 基因敲除(Mice, Knockout);心肌(Myocardium);Wnt蛋白质类(Wnt Proteins)
DOI
10.1093/hmg/ddu584
PMID
25410658
发布时间
2022-04-19
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Human molecular genetics
1704-16页
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