VEGFR3 does not sustain retinal angiogenesis without VEGFR2.

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第一作者： Georgia,Zarkada

第一单位： Wihuri Research Institute and Translational Cancer Biology Program, Biomedicum Helsinki, University of Helsinki, FIN-00014 Helsinki, Finland;

作者： Georgia,Zarkada ^[1] ; Krista,Heinolainen ^[1] ; Taija,Makinen ^[2] ; Yoshiaki,Kubota ^[3] ; Kari,Alitalo ^[4]

作者单位： Wihuri Research Institute and Translational Cancer Biology Program, Biomedicum Helsinki, University of Helsinki, FIN-00014 Helsinki, Finland; ^[1] Lymphatic Development Laboratory, Cancer Research UK London Research Institute, London WC2A 3LY, United Kingdom; Rudbeck Laboratory, Department of Immunology, Genetics, and Pathology, Uppsala University, 751 85 Uppsala, Sweden; and. ^[2] The Laboratory of Vascular Biology, School of Medicine, Keio University, Shinjuku-ku, Tokyo 160-8582, Japan. ^[3] Wihuri Research Institute and Translational Cancer Biology Program, Biomedicum Helsinki, University of Helsinki, FIN-00014 Helsinki, Finland; kari.alitalo@helsinki.fi. ^[4]

关键词 Notch VEGFC VEGFD anti-angiogenesis lymphangiogenesis

主题词动物(Animals);小鼠(Mice);新生血管化, 生理性(Neovascularization, Physiologic);视网膜血管(Retinal Vessels);血管内皮生长因子受体2(Vascular Endothelial Growth Factor Receptor-2);血管内皮生长因子受体3(Vascular Endothelial Growth Factor Receptor-3)

DOI 10.1073/pnas.1423278112

PMID 25561555

发布时间 2022-01-29

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准确一般字义偏差语意偏离译文作用小提交