换一批
换一批bcl-2 modulation of apoptosis induced by anticancer drugs: resistance to thymidylate stress is independent of classical resistance pathways.
第一作者：
T C,Fisher
第一单位：
Cancer Research Campaign Molecular and Cellular Pharmacology Group, School of Biological Sciences, University of Manchester, United Kingdom.
作者：
主题词
细胞凋亡(Apoptosis);伯基特淋巴瘤(Burkitt Lymphoma);DNA损伤(DNA Damage);DNA, 肿瘤(DNA, Neoplasm);抗药性(Drug Resistance);氟尿苷(Floxuridine);叶酸(Folic Acid);人类(Humans);原癌基因蛋白质类(Proto-Oncogene Proteins);原癌基因蛋白质c-bcl-2(Proto-Oncogene Proteins c-bcl-2);喹唑啉类(Quinazolines);胸苷酸合酶(Thymidylate Synthase);转染(Transfection);肿瘤细胞, 培养的(Tumor Cells, Cultured)
PMID
8324744
发布时间
2013-11-21
- 浏览33
Cancer research
3321-6页
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