The oncoprotein Bcl-3 can facilitate NF-kappa B-mediated transactivation by removing inhibiting p50 homodimers from select kappa B sites.
第一作者:
G,Franzoso
第一单位:
Laboratory of Immunoregulation, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892.
作者:
主题词
动物(Animals);锚蛋白类(Ankyrins);结合部位(Binding Sites);细胞核(Cell Nucleus);DNA(DNA);荧光抗体技术(Fluorescent Antibody Technique);NF-κB(NF-kappa B);原癌基因蛋白质类(Proto-Oncogene Proteins);重复序列, 核酸(Repetitive Sequences, Nucleic Acid);转录因子(Transcription Factors);转录激活(Transcriptional Activation);肿瘤细胞, 培养的(Tumor Cells, Cultured)
DOI
10.1002/j.1460-2075.1993.tb06067.x
PMID
8404857
发布时间
2022-05-11
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The EMBO journal
3893-901页
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