Retinoic acid inhibits induction of c-Jun protein by ultraviolet radiation that occurs subsequent to activation of mitogen-activated protein kinase pathways in human skin in vivo.
第一作者:
G J,Fisher
第一单位:
Department of Dermatology, University of Michigan Medical School, Ann Arbor, Michigan 48109-0609, USA. dianemch@umich.edu
作者:
主题词
转录激活因子2(Activating Transcription Factor 2);抗肿瘤药(Antineoplastic Agents);钙-钙调素依赖性蛋白激酶类(Calcium-Calmodulin-Dependent Protein Kinases);cAMP反应元件结合蛋白质(Cyclic AMP Response Element-Binding Protein);基因表达(Gene Expression);人类(Humans);JNK丝裂原活化蛋白激酶类(JNK Mitogen-Activated Protein Kinases);丝裂原激活蛋白激酶类(Mitogen-Activated Protein Kinases);神经组织蛋白质类(Nerve Tissue Proteins);启动区, 遗传(Promoter Regions, Genetic);原癌基因蛋白质c-jun(Proto-Oncogene Proteins c-jun);原癌基因蛋白质类p21(ras)(Proto-Oncogene Proteins p21(ras));信号传导(Signal Transduction);皮肤(Skin);转录因子AP-1(Transcription Factor AP-1);转录因子(Transcription Factors);转录, 遗传(Transcription, Genetic);维甲酸(Tretinoin);紫外线(Ultraviolet Rays);增量调节(Up-Regulation);p38丝裂原活化蛋白激酶类(p38 Mitogen-Activated Protein Kinases)
DOI
10.1172/JCI2153
PMID
9502786
发布时间
2018-12-01
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