换一批Cardioprotective mechanism of S-nitroso-N-acetylcysteine via S-nitrosated betadrenoceptor-2 in the LDLr-/- mice.
第一单位：
Marc and Ruti Bell Vascular Biology and Disease Program, Leon H. Charney Division of Cardiology, Department of Medicine, New York University School of Medicine, New York, NY, USA; Department of Anatomy, Cellular Biology and Physiology, State University of Campinas (UNICAMP), Biology Institute, São Paulo, Brazil. Electronic address: amarylisclaudine.wanschel@nyumc.org.
作者：
医学主题词
乙酰半胱氨酸(Acetylcysteine);动物(Animals);抗氧化剂(Antioxidants);细胞凋亡(Apoptosis);血脂异常(Dyslipidemias);内质网(Endoplasmic Reticulum);基因表达调控(Gene Expression Regulation);过氧化氢(Hydrogen Peroxide);肥大, 左心室(Hypertrophy, Left Ventricular);男(雄)性(Male);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, 基因敲除(Mice, Knockout);肌细胞, 心脏(Myocytes, Cardiac);一氧化氮合酶Ⅲ型(Nitric Oxide Synthase Type III);氮(Nitrogen);氧化性应激(Oxidative Stress);活性氧(Reactive Oxygen Species);受体, 肾上腺素能β2(Receptors, Adrenergic, beta-2);受体, LDL(Receptors, LDL);超氧化物类(Superoxides)
DOI
10.1016/j.niox.2013.12.003
PMID
24333561
发布时间
2016-10-20
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Nitric oxide
58-66页
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