Bcl-2 and Bcl-xL mediate resistance to receptor tyrosine kinase-targeted therapy in lung and gastric cancer.
第一作者:
Junfei,Jin
第一单位:
aLaboratory of Hepatobiliary and Pancreatic Surgery, Affiliated Hospital of Guilin Medical University bChina-USA Lipids in Health and Disease Research Center, Guilin Medical University, Guilin, China cDepartment of Biochemistry and Molecular Biology, Medical University of South Carolina, Charleston, South Carolina, USA.
作者:
医学主题词
苯胺化合物(Aniline Compounds);动物(Animals);抗肿瘤联合化疗方案(Antineoplastic Combined Chemotherapy Protocols);细胞系, 肿瘤(Cell Line, Tumor);抗药性, 肿瘤(Drug Resistance, Neoplasm);药物协同作用(Drug Synergism);人类(Humans);肺肿瘤(Lung Neoplasms);MAP激酶信号系统(MAP Kinase Signaling System);男(雄)性(Male);小鼠(Mice);小鼠, 裸(Mice, Nude);分子靶向治疗(Molecular Targeted Therapy);蛋白激酶抑制剂(Protein Kinase Inhibitors);原癌基因蛋白质c-akt(Proto-Oncogene Proteins c-akt);原癌基因蛋白质c-bcl-2(Proto-Oncogene Proteins c-bcl-2);随机分配(Random Allocation);胃肿瘤(Stomach Neoplasms);磺胺类(Sulfonamides);增量调节(Up-Regulation);异种移植模型抗肿瘤试验(Xenograft Model Antitumor Assays);bcl-X蛋白质(bcl-X Protein)
DOI
10.1097/CAD.0000000000000561
PMID
28885267
发布时间
2019-12-10
基金项目
UL1 TR001450/TR/NCATS NIH HHS/United States
- 浏览13
Anti-cancer drugs
1141-1149页
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