NF-κB/mTOR/MYC Axis Drives PRMT5 Protein Induction After T Cell Activation via Transcriptional and Non-transcriptional Mechanisms.
第一作者:
Lindsay M,Webb
第一单位:
Division of Medical Laboratory Science, Wexner Medical Center, School of Health and Rehabilitation Sciences, College of Medicine, The Ohio State University, Columbus, OH, United States.;Biomedical Sciences Graduate Program, The Ohio State University, Columbus, OH, United States.
作者:
主题词
动物(Animals);细胞系(Cell Line);脑脊髓炎, 自身免疫性, 实验性(Encephalomyelitis, Autoimmune, Experimental);淋巴细胞活化(Lymphocyte Activation);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, 转基因(Mice, Transgenic);多发性硬化(Multiple Sclerosis);NF-κB(NF-kappa B);蛋白质精氨酸N-甲基转移酶(Protein-Arginine N-Methyltransferases);原癌基因蛋白质c-myc(Proto-Oncogene Proteins c-myc);TOR丝氨酸-苏氨酸激酶(TOR Serine-Threonine Kinases);Th1细胞(Th1 Cells);Th2细胞(Th2 Cells);转录, 遗传(Transcription, Genetic)
DOI
10.3389/fimmu.2019.00524
PMID
30941147
发布时间
2024-11-21
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