2006年第20卷03期
共11篇1. Telomere elongation is under the control of the RNAi-based mechanism in the Drosophila germline. 345-54页
作者:Mikhail,Savitsky; Dmitry,Kwon; Pavel,Georgiev; Alla,Kalmykova; Vladimir,Gvozdev
2. A mechanism of AP-1 suppression through interaction of c-Fos with lamin A/C. 307-20页
作者:Carmen,Ivorra; Markus,Kubicek; José M,González; Silvia M,Sanz-González; Alberto,Alvarez-Barrientos; José-Enrique,O'Connor; Brian,Burke; Vicente,Andrés
3. Selective and antagonistic functions of SWI/SNF and Mi-2beta nucleosome remodeling complexes during an inflammatory response. 282-96页
作者:Vladimir R,Ramirez-Carrozzi; Aaron A,Nazarian; Caiyi C,Li; Sarah L,Gore; Rupa,Sridharan; Anthony N,Imbalzano; Stephen T,Smale
4. Coordination and communication between the p53 and IGF-1-AKT-TOR signal transduction pathways. 267-75页
作者:Arnold J,Levine; Zhaohui,Feng; Tak W,Mak; Han,You; Shengkan,Jin
5. Dual degradation signals control Gli protein stability and tumor formation. 276-81页
作者:Erik G,Huntzicker; Ivette S,Estay; Hanson,Zhen; Ludmila A,Lokteva; Peter K,Jackson; Anthony E,Oro
6. Phosphorylation-dependent maturation of Neurospora circadian clock protein from a nuclear repressor toward a cytoplasmic activator. 297-306页
作者:Tobias,Schafmeier; Krisztina,Káldi; Axel,Diernfellner; Christian,Mohr; Michael,Brunner
7. Binding of a novel SMG-1-Upf1-eRF1-eRF3 complex (SURF) to the exon junction complex triggers Upf1 phosphorylation and nonsense-mediated mRNA decay. 355-67页
作者:Isao,Kashima; Akio,Yamashita; Natsuko,Izumi; Naoyuki,Kataoka; Ryo,Morishita; Shinichi,Hoshino; Mutsuhito,Ohno; Gideon,Dreyfuss; Shigeo,Ohno
8. Sex-lethal imparts a sex-specific function to UNR by recruiting it to the msl-2 mRNA 3' UTR: translational repression for dosage compensation. 368-79页
作者:Kent,Duncan; Marica,Grskovic; Claudia,Strein; Karsten,Beckmann; Ricarda,Niggeweg; Irina,Abaza; Fátima,Gebauer; Matthias,Wilm; Matthias W,Hentze
9. Control of ribonucleotide reductase localization through an anchoring mechanism involving Wtm1. 334-44页
作者:Yang David,Lee; Stephen J,Elledge