Tumor necrosis factor alpha up-regulates non-lymphoid Fas-ligand following superantigen-induced peripheral lymphocyte activation.
第一作者:
Michael J,Pinkoski
第一单位:
Division of Cellular Immunology, La Jolla Institute for Allergy and Immunology, San Diego, California 92121, USA. michael@liai.org
作者:
医学主题词
动物(Animals);抗原, CD(Antigens, CD);细胞, 培养的(Cells, Cultured);Fas配体蛋白质(Fas Ligand Protein);肠黏膜(Intestinal Mucosa);淋巴细胞活化(Lymphocyte Activation);膜糖蛋白类(Membrane Glycoproteins);小鼠(Mice);小鼠, 近交BALB C(Mice, Inbred BALB C);小鼠, 近交C57BL(Mice, Inbred C57BL);RNA, 信使(RNA, Messenger);受体, 肿瘤坏死因子(Receptors, Tumor Necrosis Factor);受体, 肿瘤坏死因子, Ⅰ型(Receptors, Tumor Necrosis Factor, Type I);超抗原(Superantigens);肿瘤坏死因子α(Tumor Necrosis Factor-alpha);增量调节(Up-Regulation)
DOI
10.1074/jbc.M208167200
PMID
12196549
发布时间
2021-02-09
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