Cohesin defects lead to premature sister chromatid separation, kinetochore dysfunction, and spindle-assembly checkpoint activation.
第一作者:
Md Tozammel,Hoque
第一单位:
Laboratory of Cell Cycle Regulation, Department of Gene Mechanisms, Graduate School of Biostudies, Kyoto University, Kitashirakawa Oiwake-cho, Sakyo-ku, Japan.
作者:
医学主题词
细胞周期蛋白质类(Cell Cycle Proteins);染色单体(Chromatids);染色体蛋白质类, 非组蛋白(Chromosomal Proteins, Non-Histone);染色体分离(Chromosome Segregation);人类(Humans);分裂间期(Interphase);染色粒(Kinetochores);微管(Microtubules);有丝分裂(Mitosis);核蛋白质类(Nuclear Proteins);磷蛋白类(Phosphoproteins);酿酒酵母蛋白质类(Saccharomyces cerevisiae Proteins)
DOI
10.1074/jbc.M206836200
PMID
12200439
发布时间
2022-02-27
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