Reduced cardiac efficiency and altered substrate metabolism precedes the onset of hyperglycemia and contractile dysfunction in two mouse models of insulin resistance and obesity.
第一作者:
Jonathan,Buchanan
第一单位:
Program in Human Molecular Biology and Genetics, University of Utah, Salt Lake City, 84112, USA.
作者:
医学主题词
动物(Animals);糖尿病并发症(Diabetes Complications);糖尿病(Diabetes Mellitus);基因表达(Gene Expression);心脏(Heart);高血糖症(Hyperglycemia);胰岛素抗药性(Insulin Resistance);男(雄)性(Male);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);心肌收缩(Myocardial Contraction);心肌(Myocardium);肌球蛋白重链(Myosin Heavy Chains);肥胖症(Obesity);氧耗量(Oxygen Consumption);PPARα(PPAR alpha)
DOI
10.1210/en.2005-0938
PMID
16141388
发布时间
2014-11-20
- 浏览16
Endocrinology
5341-9页
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