Metallothionein alleviates cardiac contractile dysfunction induced by insulin resistance: role of Akt phosphorylation, PTB1B, PPARgamma and c-Jun.
第一作者:
C X,Fang
第一单位:
Division of Pharmaceutical Sciences & Center for Cardiovascular Research and Alternative Medicine, University of Wyoming, Laramie, WY 82071-3375, USA.
作者:
医学主题词
动物(Animals);胰岛素(Insulin);胰岛素抗药性(Insulin Resistance);JNK丝裂原活化蛋白激酶类(JNK Mitogen-Activated Protein Kinases);男(雄)性(Male);金属硫蛋白(Metallothionein);小鼠(Mice);小鼠, 近交系(Mice, Inbred Strains);小鼠, 转基因(Mice, Transgenic);心肌收缩(Myocardial Contraction);肌细胞, 心脏(Myocytes, Cardiac);癌基因蛋白质v-akt(Oncogene Protein v-akt);氧化性应激(Oxidative Stress);PPARγ(PPAR gamma);磷酰化(Phosphorylation);蛋白质酪氨酸磷酸酶, 非受体1型(Protein Tyrosine Phosphatase, Non-Receptor Type 1);蛋白质酪氨酸磷酸酶类(Protein Tyrosine Phosphatases);受体, 胰岛素(Receptor, Insulin);信号传导(Signal Transduction)
DOI
10.1007/s00125-005-1940-y
PMID
16172869
发布时间
2024-03-06
- 浏览15
Diabetologia
2412-21页
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