换一批Partial deletion of the nicotinic cholinergic receptor alpha 4 or beta 2 subunit genes changes the acetylcholine sensitivity of receptor-mediated 86Rb+ efflux in cortex and thalamus and alters relative expression of alpha 4 and beta 2 subunits.
第一作者：
Cecilia,Gotti
第一单位：
Department of Medical Pharmacology and Center of Excellence on Neurodegenerative Diseases, Institute of Neuroscience, Cellular and Molecular Pharmacology, Consiglio Nazionale delle Ricerche, University of Milan, Milan, Italy.
作者：
医学主题词
乙酰胆碱(Acetylcholine);动物(Animals);大脑皮质(Cerebral Cortex);基因缺失(Gene Deletion);基因表达调控(Gene Expression Regulation);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);蛋白质亚单位(Protein Subunits);受体, 烟碱(Receptors, Nicotinic);铷放射性同位素(Rubidium Radioisotopes);丘脑(Thalamus)
DOI
10.1124/mol.108.045203
PMID
18337473
发布时间
2025-02-13
- 浏览102
Molecular pharmacology
1796-807页
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