Deletion of protein kinase Cε in mice has limited effects on liver metabolite levels but alters fasting ketogenesis and gluconeogenesis.
第一作者:
K,Raddatz
第一单位:
Diabetes and Obesity Program, Garvan Institute of Medical Research, 384 Victoria Street, Darlinghurst, NSW, 2010, Australia.;Competence Centre Functional Genomics-Pathoproteomics, University of Greifswald, Greifswald, Germany.
作者:
医学主题词
动物(Animals);禁食(Fasting);脂肪酸类(Fatty Acids);基因缺失(Gene Deletion);糖原异生(Gluconeogenesis);止血(Hemostasis);胰岛素抗药性(Insulin Resistance);酮类(Ketones);肝(Liver);小鼠(Mice);小鼠, 基因敲除(Mice, Knockout);模型, 动物(Models, Animal);蛋白激酶Cε(Protein Kinase C-epsilon)
DOI
10.1007/s00125-012-2647-5
PMID
22814763
发布时间
2021-10-21
- 浏览12
Diabetologia
2789-2793页
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