Plasma neurofilament heavy chain levels correlate to markers of late stage disease progression and treatment response in SOD1(G93A) mice that model ALS.

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作者： Ching-Hua,Lu ^[1] ; Axel,Petzold ; Bernadett,Kalmar ; James,Dick ; Andrea,Malaspina ; Linda,Greensmith

作者单位： Sobell Department of Motor Neuroscience and Movement Disorders, MRC Centre for Neuromuscular Disorders, UCL Institute of Neurology, University College London, London, United Kingdom. ^[1]

主题词肌萎缩侧索硬化(Amyotrophic Lateral Sclerosis);动物(Animals);疾病模型, 动物(Disease Models, Animal);疾病恶化(Disease Progression);酶联免疫吸附测定(Enzyme-Linked Immunosorbent Assay);女(雌)性(Female);羟胺类(Hydroxylamines);小鼠(Mice);小鼠, 近交C57BL(Mice, Inbred C57BL);小鼠, 转基因(Mice, Transgenic);肌(Muscles);神经微丝蛋白质类(Neurofilament Proteins);磷酰化(Phosphorylation);脊髓(Spinal Cord);超氧化物歧化酶(Superoxide Dismutase);治疗结果(Treatment Outcome)

DOI 10.1371/journal.pone.0040998

PMID 22815892

发布时间 2022-03-09

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PloS one

2012年7卷7期

e40998页

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Plasma neurofilament heavy chain levels correlate to markers of late stage disease progression and treatment response in SOD1(G93A) mice that model ALS.

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Plasma neurofilament heavy chain levels correlate to markers of late stage disease progression and treatment response in SOD1(G93A) mice that model ALS.

PloS one

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PloS one

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