Chronic kidney disease-induced cardiac fibrosis is ameliorated by reducing circulating levels of a non-dialysable uremic toxin, indoxyl sulfate.

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作者： Suree,Lekawanvijit ^[1] ; Andrew R,Kompa ; Minako,Manabe ; Bing H,Wang ; Robyn G,Langham ; Fuyuhiko,Nishijima ; Darren J,Kelly ; Henry,Krum

作者单位： Centre of Cardiovascular Research and Education in Therapeutics, Department of Epidemiology and Preventive Medicine, Monash University, Melbourne, Australia. ^[1]

主题词动物(Animals);碳(Carbon);心肌疾病(Cardiomyopathies);纤维化(Fibrosis);靛甙(Indican);男(雄)性(Male);氧化物(Oxides);随机分配(Random Allocation);大鼠(Rats);大鼠, Sprague-Dawley(Rats, Sprague-Dawley);肾功能不全, 慢性(Renal Insufficiency, Chronic)

DOI 10.1371/journal.pone.0041281

PMID 22829936

发布时间 2021-10-21

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PloS one

2012年7卷7期

e41281页

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Chronic kidney disease-induced cardiac fibrosis is ameliorated by reducing circulating levels of a non-dialysable uremic toxin, indoxyl sulfate.

PloS one

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PloS one

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Chronic kidney disease-induced cardiac fibrosis is ameliorated by reducing circulating levels of a non-dialysable uremic toxin, indoxyl sulfate.

PloS one

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PloS one

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