Platelets induce apoptosis during sepsis in a contact-dependent manner that is inhibited by GPIIb/IIIa blockade.

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作者： Matthew,Sharron ^[1] ; Claire E,Hoptay ; Andrew A,Wiles ; Lindsay M,Garvin ; Mayya,Geha ; Angela S,Benton ; Kanneboyina,Nagaraju ; Robert J,Freishtat

作者单位： Division of Critical Care Medicine, Children's National Medical Center, Washington, DC, United States of America. ^[1]

主题词动物(Animals);细胞凋亡(Apoptosis);血小板(Blood Platelets);半胱氨酸天冬氨酸蛋白酶(Caspases);疾病恶化(Disease Progression);颗粒酶类(Granzymes);肺(Lung);淋巴细胞(Lymphocytes);男(雄)性(Male);小鼠(Mice);肽类(Peptides);穿孔素(Perforin);血小板聚集(Platelet Aggregation);血小板糖蛋白GPⅡb-Ⅲa复合物(Platelet Glycoprotein GPIIb-IIIa Complex);脓毒症(Sepsis);脾(Spleen)

DOI 10.1371/journal.pone.0041549

PMID 22844498

发布时间 2021-10-21

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PloS one

2012年7卷7期

e41549页

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Platelets induce apoptosis during sepsis in a contact-dependent manner that is inhibited by GPIIb/IIIa blockade.

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Platelets induce apoptosis during sepsis in a contact-dependent manner that is inhibited by GPIIb/IIIa blockade.

PloS one

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PloS one

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