R-Baclofen Reverses Cognitive Deficits and Improves Social Interactions in Two Lines of 16p11.2 Deletion Mice.
第一作者:
Laura J,Stoppel
第一单位:
The Picower Institute for Learning and Memory, Department of Brain and Cognitive Sciences, Massachusetts Institute of Technology, Cambridge, MA, USA.
作者:
医学主题词
动物(Animals);孤独性障碍(Autistic Disorder);巴氯芬(Baclofen);染色体缺失(Chromosome Deletion);染色体障碍(Chromosome Disorders);染色体, 人, 16对(Chromosomes, Human, Pair 16);认知障碍(Cognition Disorders);疾病模型, 动物(Disease Models, Animal);女(雌)性(Female);GABA-B受体激动剂(GABA-B Receptor Agonists);男(雄)性(Male);记忆障碍(Memory Disorders);小鼠, 129系(Mice, 129 Strain);小鼠, 近交C57BL(Mice, Inbred C57BL);精神治疗药物(Psychotropic Drugs);社会行为(Social Behavior);种特异性(Species Specificity)
DOI
10.1038/npp.2017.236
PMID
28984295
发布时间
2024-03-27
- 浏览21
Neuropsychopharmacology
513-524页
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