换一批BCL-2 blocks perforin-induced nuclear translocation of granzymes concomitant with protection against the nuclear events of apoptosis.
第一作者：
D A,Jans
第一单位：
Nuclear Signaling Laboratory, Division for Biochemistry and Molecular Biology, John Curtin School of Medical Research, Canberra City, Australia. David.Jans@anu.edu.au
作者：
医学主题词
动物(Animals);细胞凋亡(Apoptosis);生物转运(Biological Transport);细胞核(Cell Nucleus);DNA碎片裂(DNA Fragmentation);颗粒酶类(Granzymes);动力学(Kinetics);膜糖蛋白类(Membrane Glycoproteins);小鼠(Mice);显微镜检查, 共焦(Microscopy, Confocal);穿孔素(Perforin);成孔毒素蛋白质类(Pore Forming Cytotoxic Proteins);原癌基因蛋白质c-bcl-2(Proto-Oncogene Proteins c-bcl-2);丝氨酸内肽酶类(Serine Endopeptidases);肿瘤细胞, 培养的(Tumor Cells, Cultured)
DOI
10.1074/jbc.274.7.3953
PMID
9933585
发布时间
2021-02-09
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