换一批Cooperation of p38 and extracellular signal-regulated kinase mitogen-activated protein kinase pathways during granulocyte colony-stimulating factor-induced hemopoietic cell proliferation.
第一作者：
O,Rausch
第一单位：
Cancer Research Campaign Centre for Cell and Molecular Biology, Institute of Cancer Research, Chester Beatty Laboratories, 237 Fulham Rd., London SW3 6JB, United Kingdom.
作者：
医学主题词
动物(Animals);钙-钙调素依赖性蛋白激酶类(Calcium-Calmodulin-Dependent Protein Kinases);细胞分裂(Cell Division);细胞系(Cell Line);酶激活(Enzyme Activation);酶抑制剂(Enzyme Inhibitors);类黄酮物质(Flavonoids);粒细胞集落刺激因子(Granulocyte Colony-Stimulating Factor);造血干细胞(Hematopoietic Stem Cells);人类(Humans);咪唑类(Imidazoles);白细胞介素3(Interleukin-3);细胞内信号肽和蛋白质类(Intracellular Signaling Peptides and Proteins);JNK丝裂原活化蛋白激酶类(JNK Mitogen-Activated Protein Kinases);小鼠(Mice);丝裂原激活蛋白激酶类(Mitogen-Activated Protein Kinases);磷酰化(Phosphorylation);吡啶类(Pyridines);受体, 粒细胞集落刺激因子(Receptors, Granulocyte Colony-Stimulating Factor);酪氨酸(Tyrosine);p38丝裂原活化蛋白激酶类(p38 Mitogen-Activated Protein Kinases);ras蛋白质类(ras Proteins)
DOI
10.1074/jbc.274.7.4096
PMID
9933603
发布时间
2021-12-03
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