Different mitogen-activated protein kinase signaling pathways cooperate to regulate tumor necrosis factor alpha gene expression in T lymphocytes.
第一作者:
A,Hoffmeyer
第一单位:
Institut für Medizinische Strahlenkunde und Zellforschung (MSZ), Universität Würzburg, D-97078 Würzburg, Germany.
作者:
医学主题词
衔接蛋白质类, 信号转导(Adaptor Proteins, Signal Transducing);钙-钙调素依赖性蛋白激酶类(Calcium-Calmodulin-Dependent Protein Kinases);载体蛋白质类(Carrier Proteins);酶激活(Enzyme Activation);酶抑制剂(Enzyme Inhibitors);类黄酮物质(Flavonoids);流式细胞术(Flow Cytometry);基因表达调控(Gene Expression Regulation);人类(Humans);咪唑类(Imidazoles);离子霉素(Ionomycin);JNK丝裂原活化蛋白激酶类(JNK Mitogen-Activated Protein Kinases);丝裂原激活蛋白激酶类(Mitogen-Activated Protein Kinases);吡啶类(Pyridines);信号传导(Signal Transduction);T淋巴细胞(T-Lymphocytes);十四酰佛波乙酯(Tetradecanoylphorbol Acetate);转录, 遗传(Transcription, Genetic);肿瘤细胞, 培养的(Tumor Cells, Cultured);肿瘤坏死因子α(Tumor Necrosis Factor-alpha);p38丝裂原活化蛋白激酶类(p38 Mitogen-Activated Protein Kinases)
DOI
10.1074/jbc.274.7.4319
PMID
9933633
发布时间
2022-03-11
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