Hepatitis B virus X protein differentially activates RAS-RAF-MAPK and JNK pathways in X-transforming versus non-transforming AML12 hepatocytes.
作者:
Tarn C [1]
;
Lee S [2]
;
Hu Y ;
Ashendel C ;
Andrisani OM
作者单位:
Department of Basic Medical Sciences and Medicinal Chemistry and Molecular Pharmacology, Purdue University, West Lafayette, Indiana 47907, USA.
[1]
cli.com
[2]
期刊:
《The Journal of biological chemistry》
2001年276卷37期34671-34680页
关键词:
Cell Transformation;Neoplastic;Hepatocytes drug effects;Mitogen Activated Protein Kinases metabolism;细胞转化;肿瘤;原癌基因蛋白质c-caf类;反式作用因子
发布时间:
2013-11-21
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