换一批摘要C3肾小球病(C3G)是由于补体激活、降解及沉积紊乱而引起的补体C3在肾小球的沉积,导致肾小球损伤.研究发现循环和(或)肾小球基底膜内C3b的扩大是引起免疫紊乱的关键因素.本文结合临床及实验研究,主要探讨该病的发病机制、临床特征以及治疗.
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abstractsC3 glomerulopathy (C3G) is referred to disease which is out of control of complement activation,degradation and deposition,then leads to predominant C3 deposition in glomerular and glomerular injury.The study found that C3b amplification in the circulation and/or glomerular basement membrane were the key factors that cause immune disorders.Combined with clinical and experimental research,this paper mainly discusses the pathogenesis,clinical characteristics,and treatment of the disease.
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医学主题词
肾小球(Kidney Glomerulus)研究(Research)肾小球肾炎(Glomerulonephritis)补体激活(Complement Activation)创伤和损伤(Wounds and Injuries)
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DOI
10.3760/cma.j.issn.1008-1372.2017.01.010
发布时间
2017-03-21（万方平台首次上网日期，不代表论文的发表时间）
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