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换一批C-reactive protein down-regulates endothelial nitric oxide synthase expression and promotes apoptosis in endothelial progenitor cells through receptor for advanced glycation end-products
摘要Background C-reactive protein (CRP), the prototypic marker of inflammation, has been shown to be an independent predictor of atherosclerosis.CRP can regulate receptor for advanced glycation end-products (RAGE) expression in endothelial progenitor cells (EPCs).Endothelial nitric oxide synthase (eNOS) deficiency is a pivotal event in atherogenesis.It is believed that decreased eNOS bioactivity occurs early in atherogenesis.Therefore, we tested the hypothesis that CRP can alter eNOS expression and promote apoptosis in EPCs through RAGE.
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C反应蛋白质(C-Reactive Protein)一氧化氮合酶Ⅲ型(Nitric Oxide Synthase Type III)细胞凋亡(Apoptosis)内皮祖细胞(Endothelial Progenitor Cells)动脉粥样硬化(Atherosclerosis)
母体文献
会议名称：
第11届中国介入心脏病学大会
北京
2013年
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发布时间
2016-07-25
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