Inhibition of mitochondrial permeability transition pore opening contributes to cannabinoid type 1 receptor agonist ACEA-induced neuroprotection

导出原文传递

翻译打印收藏纠错

作者： Ma, Lei ^[1] ; Niu, Wen ^[2] ; Yang, Shuai ^[3] ; Tian, Junbin ^[4] ; Luan, Hanlin ; Cao, Ming ; Xi, Wenbin ; Tu, Weifeng ; Jia, Ji ; Lv, Jianrui

作者单位： Fourth Mil Med Univ, Dept Pathol & Pathophysiol, Xian 710032, Shaanxi, Peoples R China ^[1] Guangzhou Mil Command, Guangzhou Gen Hosp, Dept Anesthesiol, Guangzhou 510010, Guangdong, Peoples R ^[2] Xi An Jiao Tong Univ, Affiliated Hosp 2, Dept Anesthesiol, Xian 710004, Shaanxi, Peoples R China ^[3] Guangzhou Mil Command, Guangzhou Gen Hosp, Dept Neurosurg, Guangzhou 510010, Guangdong, Peoples R ^[4]

关键词 Arachidonyl-2-chloroethylamide Brain ischemic injury Mitochondrial permeability transition pore Cannabinoid CB1 receptor Neuroprotection

发布时间 2019-02-19

翻译满意度评价：

准确一般字义偏差语意偏离译文作用小提交

浏览0

Neuropharmacology

2018年135卷

211-222页

相似文献

中文期刊
外文期刊
学位论文
会议论文

检索历史清除

Inhibition of mitochondrial permeability transition pore opening contributes to cannabinoid type 1 receptor agonist ACEA-induced neuroprotection

Neuropharmacology

相似文献

Neuropharmacology

相关期刊

参考文献：
指定格式：	NoteExpress EndNote RefWorks NoteFirst

检索历史 清除

Inhibition of mitochondrial permeability transition pore opening contributes to cannabinoid type 1 receptor agonist ACEA-induced neuroprotection

Neuropharmacology

相似文献

Neuropharmacology

相关期刊

检索历史清除