Class i histone deacetylase inhibition ameliorates social cognition and cell adhesion molecule plasticity deficits in a rodent model of autism spectrum disorder

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作者： Foley,A.G. ^[1] ; Gannon,S. ^[2] ; Rombach-Mullan,N. ; Prendergast,A. ; Barry,C. ; Cassidy,A.W. ; Regan,C.M.

作者单位： School of Biomolecular and Biomedical Science, UCD Conway Institute, University College Dublin ^[1] Berand Neuropharmacology, NovaUCD, Belfield Innovation Park, University College Dublin, Dublin 4 ^[2]

关键词 Biological motion Histone deacetylase MS-275 NCAM PSA SAHA Social interaction Spatial learning Synaptic plasticity

发布时间 2013-11-20

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Neuropharmacology

2012年63卷4期

750-760页

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Class i histone deacetylase inhibition ameliorates social cognition and cell adhesion molecule plasticity deficits in a rodent model of autism spectrum disorder

Neuropharmacology

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Neuropharmacology

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Class i histone deacetylase inhibition ameliorates social cognition and cell adhesion molecule plasticity deficits in a rodent model of autism spectrum disorder

Neuropharmacology

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Neuropharmacology

相关期刊

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