首页> The Journal of Steroid Biochemistry and Molecular Biology> Overexpression of aldo-keto reductase 1C3 (AKR1C3) in LNCaP cells diverts androgen metabolism towards testosterone resulting in resistance to the 5α-reductase inhibitor finasteride

Overexpression of aldo-keto reductase 1C3 (AKR1C3) in LNCaP cells diverts androgen metabolism towards testosterone resulting in resistance to the 5α-reductase inhibitor finasteride

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第一作者： Byrns,M.C.

作者： Byrns,M.C. ^[1] ; Mindnich,R. ; Duan,L. ; Penning,T.M.

作者单位： Department of Pharmacology, Perelman School of Medicine, University of Pennsylvania, 3620 Hamilton ^[1]

关键词 4-Androstene-3 17-dione 5α-Reductase Castrate resistant prostate cancer Indomethacin Type 5 17β-hydroxysteroid dehydrogenase

发布时间 2013-11-20

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The Journal of Steroid Biochemistry and Molecular Biology

2012年130卷1/2期

7-15页

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The Journal of Steroid Biochemistry and Molecular Biology

2012年130卷1/2期

7-15页

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Overexpression of aldo-keto reductase 1C3 (AKR1C3) in LNCaP cells diverts androgen metabolism towards testosterone resulting in resistance to the 5α-reductase inhibitor finasteride

The Journal of Steroid Biochemistry and Molecular Biology

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The Journal of Steroid Biochemistry and Molecular Biology

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Overexpression of aldo-keto reductase 1C3 (AKR1C3) in LNCaP cells diverts androgen metabolism towards testosterone resulting in resistance to the 5α-reductase inhibitor finasteride

The Journal of Steroid Biochemistry and Molecular Biology

相似文献

同项目论文

The Journal of Steroid Biochemistry and Molecular Biology

相关期刊

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