6-OHDA-induced apoptosis and mitochondrial dysfunction are mediated by early modulation of intracellular signals and interaction of Nrf2 and NF-??B factors

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作者： Tob贸n-Velasco,J.C. ^[1] ; Lim贸n-Pacheco,J.H. ^[2] ; Orozco-Ibarra,M. ^[3] ; Mac铆as-Silva,M. ^[4] ; V谩zquez-Victorio,G. ^[5] ; Cuevas,E. ^[6] ; Ali,S.F. ; Cuadrado,A. ; Pedraza-Chaverr铆,J. ; Santamar铆a,A.

作者单位： Departamento de Bioqu铆mica, Instituto de Investigaciones Biom茅dicas Alberto Sols UAM-CSIC, Centro ^[1] Division of Neurotoxicology, National Center for Toxicological Research - FDA, Jefferson, AR ^[2] Laboratorio de Neurobiolog铆a Molecular y Celular INNN-UNAM, Instituto Nacional de Neurolog铆a y ^[3] Departamento de Biolog铆a Celular y Desarrollo, Instituto de Fisiolog铆a Celular, Universidad ^[4] Laboratorio de Amino谩cidos Excitadores, Instituto Nacional de Neurolog铆a y Neurocirug铆a - S.S.A., ^[5] Departamento de Biolog铆a, Facultad de Qu铆mica, Universidad Nacional Aut贸noma de M茅xico, Mexico City ^[6]

关键词 6-Hydroxydopamine Apoptosis Kinases signaling Mitochondrial dysfunction Nrf2/NF-??B pathways

发布时间 2013-11-20

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Toxicology

2013年304卷

109-119页

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6-OHDA-induced apoptosis and mitochondrial dysfunction are mediated by early modulation of intracellular signals and interaction of Nrf2 and NF-??B factors

Toxicology

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6-OHDA-induced apoptosis and mitochondrial dysfunction are mediated by early modulation of intracellular signals and interaction of Nrf2 and NF-??B factors

Toxicology

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Toxicology

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