Insulin acts as an atypical KCNQ1/KCNE1‐current activator and reverses long QT in insulin‐resistant aged rats by accelerating the ventricular action potential repolarization through affecting the β3‐adrenergic receptor signaling pathway-论文-万方医学网

首页> > Insulin acts as an atypical KCNQ1/KCNE1‐current activator and reverses long QT in insulin‐resistant aged rats by accelerating the ventricular action potential repolarization through affecting the β3‐adrenergic receptor signaling pathway

Insulin acts as an atypical KCNQ1/KCNE1‐current activator and reverses long QT in insulin‐resistant aged rats by accelerating the ventricular action potential repolarization through affecting the β3‐adrenergic receptor signaling pathway

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第一作者： Yusuf Olgar

作者： Yusuf Olgar ^[1] ; Aysegul Durak ^[2] ; Ceylan V. Bitirim ; Erkan Tuncay ; Belma Turan

作者单位： Stem Cell Institute,Ankara University ^[1] Department of Biophysics, Faculty of Medicine,Ankara University ^[2]

关键词 ATP dependent K+‐channels cardiomyocytes electrical activity heart dysfunction insulin resistance intracellular ionic homeostasis voltage‐dependent ionic channels

发布时间 2022-04-21

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Insulin acts as an atypical KCNQ1/KCNE1‐current activator and reverses long QT in insulin‐resistant aged rats by accelerating the ventricular action potential repolarization through affecting the β3‐adrenergic receptor signaling pathway

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Insulin acts as an atypical KCNQ1/KCNE1‐current activator and reverses long QT in insulin‐resistant aged rats by accelerating the ventricular action potential repolarization through affecting the β3‐adrenergic receptor signaling pathway

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同项目论文

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