Se-(Methyl)-selenocysteine ameliorates blood-brain barrier disruption of focal cerebral ischemia mice via ferroptosis inhibition and tight junction upregulation in an Akt/GSK3β-dependent manner-论文-万方医学网

Se-(Methyl)-selenocysteine ameliorates blood-brain barrier disruption of focal cerebral ischemia mice via ferroptosis inhibition and tight junction upregulation in an Akt/GSK3β-dependent manner

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作者： Fei, Yuxiang ^[1] ; Li, Tao ; Wu, Ruoyu ; Xu, Xuejiao ; Hu, Sheng ; Yang, Ya ; Jin, Chenchen ; Tang, Wenlian ; Zhang, Xu ; Du, Qianming ; Liu, Chao

作者单位： Univ Hlth CtrNorthwestern Feinberg Sch Med Northwestern University Feinberg School of Medicine Ctr ^[1]

关键词 Ischemic stroke Se-(methyl)-selenocysteine Blood-brain barrier Tight junction proteins Ferroptosis SELENIUM SELENOPROTEINS THROMBOLYSIS INTEGRITY

发布时间 2024-04-09

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Se-(Methyl)-selenocysteine ameliorates blood-brain barrier disruption of focal cerebral ischemia mice via ferroptosis inhibition and tight junction upregulation in an Akt/GSK3β-dependent manner

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Se-(Methyl)-selenocysteine ameliorates blood-brain barrier disruption of focal cerebral ischemia mice via ferroptosis inhibition and tight junction upregulation in an Akt/GSK3β-dependent manner

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