首页> Molecular pharmacology> Kavalactones protect neural cells against amyloid beta peptide-induced neurotoxicity via extracellular signal-regulated kinase 1/2-dependent nuclear factor erythroid 2-related factor 2 activation.

Kavalactones protect neural cells against amyloid beta peptide-induced neurotoxicity via extracellular signal-regulated kinase 1/2-dependent nuclear factor erythroid 2-related factor 2 activation.

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第一作者： Wruck CJ

作者： Wruck CJ ^[1] ; Gotz ME ; Herdegen T ; Varoga D ; Brandenburg LO ; Pufe T

作者单位： Department of Anatomy and Cell Biology, RWTH Aachen, Wendlingweg 2, 52074 Aachen, Germany. cwruck@ukaachen.de ^[1]

关键词 Amyloid beta-Protein Animals Dose-Response Relationship Drug Lactones 淀粉样β蛋白动物剂量效应关系药物内酯类神经元神经保护药 PC12细胞

发布时间 2013-11-20

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Molecular pharmacology

2008年73卷6期

1785-1795页

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Molecular pharmacology

2008年73卷6期

1785-1795页

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Kavalactones protect neural cells against amyloid beta peptide-induced neurotoxicity via extracellular signal-regulated kinase 1/2-dependent nuclear factor erythroid 2-related factor 2 activation.

Molecular pharmacology

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Molecular pharmacology

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Kavalactones protect neural cells against amyloid beta peptide-induced neurotoxicity via extracellular signal-regulated kinase 1/2-dependent nuclear factor erythroid 2-related factor 2 activation.

Molecular pharmacology

相似文献

同项目论文

Molecular pharmacology

相关期刊

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