Differential phosphoproteome profiling reveals a functional role for VASP in Helicobacter pylori-induced cytoskeleton turnover in gastric epithelial cells

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作者： Knauer O ^[1] ; Binai NA ^[2] ; Carra G ^[3] ; Beckhaus T ^[4] ; Hanschmann KM ^[5] ; Renne T ^[6] ; Backert S ^[7] ; Karas M ^[8] ; Wessler S ^[9]

作者单位： [Backert, S] Otto Von Guericke Univ, Dept Med Microbiol, D-39120 Magdeburg, Germany ^[1] Carra, G ^[2] Binai, NA ^[3] Wessler, S] Paul Ehrlich Inst, Jr Res Grp, D-63225 Langen, Germany ^[4] [Knauer, O ^[5] Hanschmann, KM ^[6] [Renne, T] Univ Wurzburg, Inst Clin Biochem & Pathobiochem, Wurzburg, Germany ^[7] Karas, M] Univ Frankfurt, Inst Pharmaceut Chem, Bioctr, Frankfurt, Germany ^[8] [Beckhaus, T ^[9]

关键词 VASODILATOR-STIMULATED PHOSPHOPROTEIN CAG PATHOGENICITY ISLAND PROTEIN-KINASE PHOSPHORYLATION ACTIN-BASED MOTILITY KAPPA-B ACTIVATION FOCAL ADHESION ENA/VASP PROTEINS HUMAN PLATELETS IV SECRETION IN-VITRO

发布时间 2013-11-20

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