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BCAT1 promotes cell proliferation through amino acid catabolism in gliomas carrying wild-type IDH1

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第一作者: T?njes,M.
作者单位: Core Facility, Molecular Structural Analysis, DKFZ, Heidelberg, Germany [1] Division of Molecular Genetics, German Cancer Research Center (DKFZ), Heidelberg, Germany [2] Division of Molecular Genetics, German Cancer Research Center (DKFZ), Heidelberg, Germany, Division [3] Department of Neuropathology, Heinrich Heine University, Düsseldorf, Germany [4] Human Oncology and Pathogenesis Program, Memorial Sloan-Kettering Cancer Center, New York, NY [5] Department of Bioinformatics and Functional Genomics, Institute of Pharmacy and Molecular [6] Department of Human Nutrition, Foods and Exercise, Virginia Polytechnic Institute, Blacksburg, VA [7] Department of General Pediatrics, Division of Inborn Metabolic Diseases, University Childrens [8] Division of Epigenomics and Cancer Risk Factors, DKFZ, Heidelberg, Germany, Department of [9] Clinical Cooperation Unit Neurooncology, DKFZ, Heidelberg, Germany [10] Division of Epigenomics and Cancer Risk Factors, DKFZ, Heidelberg, Germany [11] Department of Neuropathology, Heinrich Heine University, Düsseldorf, Germany, German Cancer [12] Department of New Materials and Biosystems, Max Planck Institute for Intelligent Systems, Stuttgart [13] Division of Experimental Neurosurgery, University of Heidelberg, Heidelberg, Germany, Department of [14] Core Facility, Small Animal Imaging Center, DKFZ, Heidelberg, Germany [15]
发布时间 2013-11-20
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