GSK3 beta is involved in promoting Alzheimer's disease pathologies following chronic systemic exposure to Porphyromonas gingivalis lipopolysaccharide in amyloid precursor protein(NL-F/NL-F) knock-in mice

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作者： Jiang, Muzhou ^[1] ; Zhang, Xinwen ^[2] ; Yan, Xu ^[3] ; Mizutani, Shinsuke ^[4] ; Kashiwazaki, Haruhiko ; Ni, Junjun ; Wu, Zhou

作者单位： Fac Dent Sci,Kyushu Univ ^[1] Ctr Implant Dent,China Med Univ ^[2] Sch Stomatol,China Med Univ ^[3] Sch Life Sci,Beijing Inst Technol ^[4]

关键词 PgLPS Microglia Neuroinflammation Tau hyperphosphorylation GSK3 beta TNF-alpha Alzheimer 's disease MILD COGNITIVE IMPAIRMENT TAU PATHOLOGY INFLAMMATION BETA PHOSPHORYLATION MICROGLIA DEFICITS MEMORY GSK3 NEUROTOXICITY

发布时间 2023-10-21

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Brain, Behavior, and Immunity

2021年98卷

1-12页

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GSK3 beta is involved in promoting Alzheimer's disease pathologies following chronic systemic exposure to Porphyromonas gingivalis lipopolysaccharide in amyloid precursor protein(NL-F/NL-F) knock-in mice

Brain, Behavior, and Immunity

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Brain, Behavior, and Immunity

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GSK3 beta is involved in promoting Alzheimer's disease pathologies following chronic systemic exposure to Porphyromonas gingivalis lipopolysaccharide in amyloid precursor protein(NL-F/NL-F) knock-in mice

Brain, Behavior, and Immunity

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Brain, Behavior, and Immunity

相关期刊

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