Helicobacter pylori CagA Containing ITAM-Like Sequences Localized to Lipid Rafts Negatively Regulates VacA-Induced Signaling In Vivo

导出原文传递

翻译打印收藏纠错

作者： Momoyo Asahi ^[1] ; Yukie Tanaka ^[2] ; Toshimasa Izumi ^[3] ; Yoshiyuki Ito ^[4] ; Hironobu Naiki ^[5] ; Dangeruta Kersulyte ^[6] ; Kazutake Tsujikawa ^[7] ; Masakazu Saito ^[8] ; Shigeur Yanagi ^[9]

作者单位： Department of molecular micrbiology, Washington University School of Medicine, St. Louis, 63110, USA ^[1] Department of Genome Sciences, Kobe University Graduate School of Medicine, Kobe, 650-0017, Japan ^[2] Fukui General Hospital, Fukui, 910-8561, Japan ^[3] Faculty of Nursing and Social Welfare Science, Fukui Prefectural University, Fukui 910-1195, Japan ^[4] Department of Internal Medicine Fukui Medical University, Fukui, 910-1193, Japan ^[5] Faculty of Nursing and Social Welfare Science, Fukui Prefectural University, Fukui 910-1195, JapanDepartment of Genome Sciences, Kobe University Graduate School of Medicine, Kobe, 650-0017, Japan ^[6] Department of Immunology Graduate School of Pharmaceutical Sciences Osaka University, Osaka, 565-0871, Japan ^[7] Department of Pathology, Fukui Medical University, Fukui, 910-1193, Japan ^[8] Central Research Laboratories Fukui Medical University, Fukui, 910-1193, Japan ^[9]

关键词 Helicobacter pylori CagA immuno-receptor tyrosine-based activation motif (ITAM)-like sequences G protein-coupled receptor kinase interactor 1 (GIT1)/Cool-associated tyrosine-phosphorylated 1 (Cat1)CagA-induced vesicle

发布时间 2013-11-20

翻译满意度评价：

准确一般字义偏差语意偏离译文作用小提交