Conditional knockout of CRMP2 in neurons, but not astrocytes, disrupts spinal nociceptive neurotransmission to control the initiation and maintenance of chronic neuropathic pain-论文-万方医学网

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Conditional knockout of CRMP2 in neurons, but not astrocytes, disrupts spinal nociceptive neurotransmission to control the initiation and maintenance of chronic neuropathic pain

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第一作者： Boinon, Lisa

作者： Boinon, Lisa ^[1] ; Yu, Jie ^[2] ; Madura, Cynthia L. ; Chefdeville, Aude ; Feinstein, Douglas L. ; Moutal, Aubin ; Khanna, Rajesh

作者单位： Coll Med,Univ Arizona ^[1] Dept Anesthesiol,Univ Illinois ^[2]

关键词 conditional knockout synaptic transmission mechanical allodynia CRMP2 LONG-TERM POTENTIATION PROTEIN-KINASE-II DORSAL-HORN PRESYNAPTIC INHIBITION CENTRAL SENSITIZATION SYNAPTIC CURRENTS MODEL HYPERSENSITIVITY PHOSPHORYLATION EXCITABILITY

发布时间 2022-04-21

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Conditional knockout of CRMP2 in neurons, but not astrocytes, disrupts spinal nociceptive neurotransmission to control the initiation and maintenance of chronic neuropathic pain

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Conditional knockout of CRMP2 in neurons, but not astrocytes, disrupts spinal nociceptive neurotransmission to control the initiation and maintenance of chronic neuropathic pain

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