The proteasome inhibitor lactacystin enhances GSH synthesis capacity by increased expression of antioxidant components in an Nrf2-independent, but p38 MAPK-dependent manner in rat colorectal carcinoma cells

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作者： Huseby, Nils-Erik ^[1] ; Ravuri, Chandra ^[2] ; Moens, Ugo

作者单位： Univ Tromso, Fac Hlth Sci, Dept Med Biol, Tumor Biol Res Grp, N-9037 Tromso, Norway ^[1] Univ Tromso, Fac Hlth Sci, Dept Med Biol, Mol Inflammat Res Grp, N-9037 Tromso, Norway ^[2]

关键词 Cystine/glutamate transporter gamma-glutamyl-transferase glutamate cysteine ligase glutathione regulation oxidative stress proteasomal inhibitors

发布时间 2019-09-05

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Free radical research

2016年50卷1/12期

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The proteasome inhibitor lactacystin enhances GSH synthesis capacity by increased expression of antioxidant components in an Nrf2-independent, but p38 MAPK-dependent manner in rat colorectal carcinoma cells

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Free radical research

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The proteasome inhibitor lactacystin enhances GSH synthesis capacity by increased expression of antioxidant components in an Nrf2-independent, but p38 MAPK-dependent manner in rat colorectal carcinoma cells

Free radical research

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Free radical research

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