The Brucella abortus xthA-1 gene product participates in base excision repair and resistance to oxidative killing but is not required for wild-type virulence in the mouse model
第一作者:
Hornback ML
作者:
关键词
AnimalsBacterial Proteins physiologyBrucella abortus drug effects enzymology genetics pathogenicityBrucellosis microbiologyDNA DamageDNA RepairDNABacterialEnzyme Inhibitors pharmacologyExodeoxyribonucleases physiologyFemaleHydrogen Peroxide pharmacologyMethyl Methanesulfonate pharmacologyMiceInbred BALB CMolsidomine analogs &derivatives pharmacologyMutagens pharmacologyOxidants pharmacologyResearch SupportN.I.H.ExtramuralVirulence
发布时间
2013-12-20
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Journal of bacteriology
1295-1300页
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