Diminished Acyl-CoA synthetase isoform 4 activity in INS 832/13 cells reduces cellular epoxyeicosatrienoic acid levels and results in impaired glucose-stimulated insulin secretion

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作者： Klett,E.L. ^[1] ; Chen,S. ^[2] ; Edin,M.L. ^[3] ; Li,L.O. ^[4] ; Ilkayeva,O. ; Zeldin,D.C. ; Newgard,C.B. ; Coleman,R.A.

作者单位： Division of Intramural Research, NIEHS, National Institutes of Health, Research Triangle Park, NC ^[1] Department of Nutrition, University of North Carolina School of Public Health, Chapel Hill, NC ^[2] Department of Medicine, Division of Endocrinology, University of North Carolina School of Medicine ^[3] Sarah W. Stedman Nutrition and Metabolism Center, Department of Pharmacology and Cancer Biology ^[4]

发布时间 2014-01-21

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The Journal of biological chemistry

2013年288卷30期

21618-21629页

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Diminished Acyl-CoA synthetase isoform 4 activity in INS 832/13 cells reduces cellular epoxyeicosatrienoic acid levels and results in impaired glucose-stimulated insulin secretion

The Journal of biological chemistry

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The Journal of biological chemistry

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Diminished Acyl-CoA synthetase isoform 4 activity in INS 832/13 cells reduces cellular epoxyeicosatrienoic acid levels and results in impaired glucose-stimulated insulin secretion

The Journal of biological chemistry

相似文献

The Journal of biological chemistry

相关期刊

检索历史清除